Varicella
The
Varicella vaccine is given at twelve months. In today’s modern world
childhood infections have very few consequences. Actually having the
diseases can impart lifetime immunity whereas vaccine immunity is only
temporary. For example this vaccine has an effective rate estimated at six
to ten years. If effective it will postpone
chicken pox until
adulthood when death from the disease is twenty times more likely. Merck’s
own literature states “Further duration of protection is unknown at present
and the need for booster is not defined.” This vaccine was licensed in 1995
so no long-term studies have been done. Some healthcare professionals are
concerned that the ‘herpes viruses’ could reactivate later in life in the
form of shingles or Epstein Barr.
The Illinois State Board of health voted
against the Chickenpox vaccine mandate due to nine thousand adverse
reactions reported in 1995. As of August 2000, Illinois had 231 compensation
claims had been filed for 45 deaths and 186 injuries. This vaccine is made
from fetal bovine serum, aborted fetal tissue cells and Monosodium
Glutamate.
The truth is no one knows the risks of
injecting mutated DNA
into children, but what has
been reported is Bell’s Palsy, encephalitis and seizures just to name a
few. This vaccine is now licensed and recommended by health authorities.
According to a new report, during an outbreak of chickenpox in Minnesota
in the fall of 2002, more than half the children who became infected had
been immunized with the varicella vaccine. What this says to me is
this vaccine is not working. What this says to the
FDA is we need another
dose.
Here is an article by Dan Olmstead about Chicken pox.
The Age of Autism: Anna's Last Days -- 1
By Dan Olmsted for UPI. http://tinyurl.com/gp3ch
On April 26 a Scottish child named Anna Duncan attended a party where
two children had chickenpox. Nine days later she got her routine
measles-mumps-rubella vaccination. Four days after that she developed
classic chickenpox symptoms -- spots and fever. One week later, on May 14,
Anna was dead from an apparent seizure. She was 17 months old. Now her
father, John, is struggling with the sudden loss of a bright, lively child
-- and increasingly suspicious that the MMR shot during an apparent
chickenpox infection triggered her death. Those suspicions deepened after he
came across Age of Autism's recent investigative series, Pox, which found
that giving MMR and chickenpox vaccines at the same time might raise the
risk of autism in a susceptible subset of children. By happenstance, the
series began the week before Anna's exposure to chickenpox and ended the
week after her death.
In Anna's case, Duncan believes the chickenpox she caught at the party
suppressed her immune system to the point that the measles virus from the
MMR triggered a fatal seizure. "I feel now that I have an answer to our
daughter's death," said Duncan, of Cardrona, Scotland. "What I'm going to
try to do with this is force a fatal accident inquiry, because there is a
potential scenario here where it could happen again, and if (they) realize
that this is a developing story, it can only get bigger." The Pox
series centered on several autistic children in Olympia, Wash., whose
families had problematic histories with chickenpox and related herpesviruses.
All of the children got the MMR and chickenpox vaccines, in most cases at
their 12-month checkups; two of the children were in Merck &Co. clinical
trials of investigational chickenpox vaccines in combinationwith the MMR.
John Duncan said that like the Olympia families, he also had unusual
reactions to viral infections and experienced a monthlong outbreak of
pox-like spots just after Anna was born. He took photographs at the time to
document the spots, which spread diffusely from his abdomen. "I believe her
response to the MMR while infected with chickenpox was due to her genetic
makeup from myself," Duncan wrote in a posting on the British Web site
jabs.org.uk. "Anna's normal response to a benign childhood illness, for
which recovery was a formality, was interrupted by the MMR vaccine, which
due to her understandable immunosuppression resulted in the replication of
the measles virus -- 'virus replication,' an accepted and understood medical
event in relation to vaccines."
It will be weeks before laboratory tests confirm whether Anna had
chickenpox and health authorities rule on cause of death. But authorities in
both Britain and the United States assert there is no association between
the vaccines and serious health problems. They say the real risk is
foregoing vaccinations based on unfounded fears. The Daily Mail
reported in June that "Britain is now in the grip of the biggest measles
outbreak since the vaccine's introduction in 1988. Doctors have reported
hundreds of cases of measles since January in just three areas of the
country, including the death of a 13-year-old boy." Last week "a group
of Britain's leading pediatricians and childhood vaccination experts ...
warned that more children will die unless a line is drawn under the autism
and MMR vaccine controversy," according to Britain's Guardian newspaper. "In
an open letter, 30 scientists, including some of the country's most eminent
child health experts, say that an overwhelming body of evidence shows the
vaccine is safe. They add that urgent immunizations are necessary to prevent
potentially devastating outbreaks among schoolchildren." The MMR vaccine
Anna received was Priorix, manufactured by GlaxoSmithKline. Chickenpox
vaccine is not routinely administered in Britain; in the United States it is
recommended by health authorities for all children beginning at age 12
months.
John Duncan provided this sequence of events leading up to Anna's
death.
Wednesday, April 26 -- Anna attended the party with her mother,
Veronica, where one child was getting over chickenpox and that child's
younger sister had all the symptoms of chickenpox.
Friday, May 5 -- Anna got her MMR shot at Haylodge Health Centre,
Peebles, Scottish Borders; her mother questioned whether Anna's runny nose
and exposure to chickenpox was a cause for concern. The healthcare worker
said it was not.
Tuesday, May 9 -- Anna developed signs of chickenpox with spots
appearing and a slight fever. This developed into what appeared to be
classic chickenpox.
Sunday, May 14 -- Anna died around 9 a.m. with what appeared to be a
seizure, with evidence of blood on her lips and on sheets in close proximity
to her mouth. "When Anna had chickenpox we gave her (a fever reducer)
to bring her temperature down when it spiked," John Duncan said. "Her
temperature according to her mother, who is a nurse, seemed to stabilize on
the Saturday night through to Sunday morning, but Anna became restless early
on Sunday morning and had two very smelly nappy (diaper) changes. A tired
mother put Anna in her cot at around 6 p.m. as she seemed to be more
contented on her own. "Anna's death came as a major shock to us all because
at no time did we think that she was going to die. The seizure would have
been undetectable in the circumstances. I was with (son) Cameron that
morning downstairs because I thought Anna had turned the corner." Duncan
said a doctor who came to the house to confirm the death told his wife it
appeared "Anna had chickenpox." She may also have started developing new
spots characteristic of measles, he said.
"I would say at time of death there were more measles-like spots
appearing around her neck. But I cannot be too sure." Duncan asked on
the Jabs site: "Could this scenario cause autism? Is there a genetic
susceptibility in some children to deal with the herpesvirus in a different
way to the normal response, making these children more at risk to a bad
reaction from MMR at the time of herpes infection? ... "Had Anna
survived her bout of seizure 10 days after her MMR, her brain very possibly
could have been damaged and a diagnosis of autism eventually given."
-- Next: Chickenpox and measles -- a troubling combination.
So, is it really a good idea to inject virus into humans? Take a look at
this study.
PMID: 15603537 [PubMed - indexed for MEDLINE]
Brain Pathol. 2006 Jan;16(1):1-14.
Metallothioneins and zinc dysregulation contribute to neurodevelopmental
damage in a model of perinatal viral infection.
* Williams BL. et al Greene Infectious Disease Laboratory, Mailman
School of Public Health, Columbia University, New York, NY 10032, USA.
Neonatal Borna disease (NBD) virus infection in the Lewis rat results in
life-long viral persistence and causes behavioral and neurodevelopmental
abnormalities. A hallmark of the disorder is progressive loss of cerebellar
Purkinje and dentate gyrus granule cells. Findings of increased brain
metallothionein-I and -II (MT-I/-II) mRNA expression in cDNA microarray
experiments led us to investigate MT isoforms and their relationship to
brain zinc metabolism, cellular toxicity, and neurodevelopmental
abnormalities in this model. Real-time PCR confirmed marked induction of
MT-I/-II mRNA expression in the brains of NBD rats (40.5-fold increase in
cerebellum, p<0.0001; 6.8-fold increase in hippocampus, p=0.003; and
9.5-fold increase in striatum, p=0.0012), whereas a trend toward decreased
MT-III mRNA was found in hippocampus (1.25-fold decrease, p=0.0841). Double
label immunofluorescence revealed prominent MT-I/-II expression in
astrocytes throughout the brain; MT-III protein was decreased in granule
cell neurons and increased in astrocytes, with differential subcellular
distribution from cytoplasmic to nuclear compartments in NBD rat
hippocampus. Modified Timm staining of hippocampus revealed reduced zinc in
mossy fiber projections to the hilus and CA3, accumulation of zinc in glial
cells and degenerating granule cell somata, and robust mossy fiber sprouting
into the inner molecular layer of the dentate gyrus. Zinc Transporter 3
(ZnT-3) mRNA expression was decreased in hippocampus (2.3-fold decrease, p=
0.0065); staining for its correlate protein was reduced in hippocampal mossy
fibers. Furthermore, 2 molecules implicated in axonal pathfinding and mossy
fiber sprouting, the extracellular matrix glycoprotein, tenascin-R (TN-R),
and the hyaluronan receptor CD44, were increased in NBD hippocampal
neuropil. Abnormal zinc metabolism and mechanisms of neuroplasticity may
contribute to the pathogenesis of disease in this model, raising more
general implications for neurodevelopmental damage following viral
infections in early life.
PMID: 16612977 [PubMed - indexed for MEDLINE]
Typhoid fever
This vaccine is a travel vaccine and not on the standard schedule. This is
an interesting study to take a look at.Clin Infect Dis. 2004 Mar
15;38(6):771-9. Epub 2004 Feb 26. Related Articles,
Links
Postmarketing safety surveillance for
typhoid fever vaccines from the
Vaccine Adverse Event Reporting System, July 1990 through June 2002. Begier EM, Burwen DR, Haber P, Ball R; Vaccine Adverse Event Reporting
System .
Vaccine Safety Branch, Division of Epidemiology, Office of Biostatistics
and Epidemiology, Center for Biologics Evaluation and Research, Food and
Drug Administration, Rockville, Maryland 20852-1448, USA.
Vaccines against Salmonella enterica serotype Typhi are used for
prophylaxis of international travelers and have potential use as
counterbioterrorism agents. The Vaccine Adverse Event Reporting System (VAERS)
cannot usually establish causal relationships between vaccines and
reported adverse events without further research but has successfully
detected unrecognized side effects of vaccine. We reviewed reports to
VAERS for US-licensed typhoid fever vaccines for the period of July 1990
through June 2002. We received 321 reports for parenteral Vi capsular
polysaccharide vaccine and 345 reports for live, oral, attenuated Ty21a
vaccine, with 7.5% and 5.5%, respectively, describing death,
hospitalization, permanent disability, or life-threatening illness.
Unexpected frequently reported symptoms included dizziness and pruritus
for Vi vaccine and fatigue and myalgia for Ty21a vaccine.
Gastroenteritis-like illness after receipt of Ty21a vaccine and abdominal
pain after receipt of Vi vaccine, which are previously recognized events,
occasionally required hospitalization. Nonfatal anaphylaxis was reported
after both vaccines. VAERS reports do not indicate any unexpected serious
side effects that compromise these vaccines' use for travelers'
prophylaxis.
PMID: 14999618 [PubMed - in process
Yellow fever Vaccine
This vaccine is not on the recommended schedule however, if traveling to
some continents it is recommended. I recently acquired an empty vial of
yellow fever vaccine and was startled at what was written on the label.
"Avian leukosis free". Is this an admission that it one time the vaccine
did contain avian leukosis? How can we be sure its out now? Here is a
picture of the vial. Hopefully you can see it.
This is from the CDC: Yellow fever is
a mosquito-borne viral disease. Illness ranges in severity from an
influenza-like syndrome to severe hepatitis and hemorrhagic fever. Yellow
fever is caused by a zoonotic virus that is maintained in nature by
transmission between nonhuman primates and mosquito vectors. In some
situations, humans may serve as the primary host in the transmission
cycle (“urban yellow fever”). (Hmmm shedding vaccine?)
Here is one study done on the vaccine.
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&
dopt=Abstract&list_uids=15149765
Vaccine. 2004 Jun 2;22(17-18):2103-5. Related Articles,Links
Viscerotropic and neurotropic disease following vaccination with the 17D
yellow fever vaccine, ARILVAX((R)).
Kitchener S.
C/- Peterhouse Technology Park, 100 Fulbourn Road, Cambridge CBI 9PT, UK.
Yellow fever vaccine associated viscerotropic (YFV-AVD) and neurotropic (YFV-AND)
diseases have been recently identified in various countries. Previously
post-vaccination multiple organ system failure was recognised as a rare
serious adverse event of yellow fever vaccination and 21 cases of post-vaccinal
(YFV) encephalitis had been recorded. Incidence data is not available. On
investigation of vaccine surveillance reports from Europe following
distribution of more than 3 million doses of ARILVAX trade mark, four
cases each of YFV-AVD and YFV-AND were found (each 1.3 cases per million
doses distributed) for the period 1991 to 2003. The incidence for each is
higher after 1996 (2.5 cases per million doses distributed). The
incidence of these adverse events appears to be very low with ARILVAX
trade mark. Similar incidence data is required from other countries for
comparison.
Publication Types:
Letter
PMID: 15149765 [PubMed - in process]
Here is more:
http://www.eurekalert.org/pub_releases/2004-06/nymc-ric061704.php
Contact: Donna E. Moriarty
donna_moriarty@nymc.edu
914-594-4536
New York Medical College
Researcher issues caution on live virus vaccines
Vaccine flaviviruses can recombine, resulting in a new microbe with
potentially undesirable properties
VALHALLA, N.Y., June 18, 2004--A New York Medical College microbiologist
warns that live virus vaccines to prevent infectious diseases like West
Nile virus and yellow fever could have dire consequences. Should one of
the vaccine flaviviruses (Flaviviruses virions are spherical, enveloped,
and 40-50 nm in diameter. Flavi contains a linear, plus sense,
single-stranded RNA genome. There are about 70 recognized diseases in the
Flavi family. 13 cause disease in humans, such as Yellow Fever, Dengue,
and Japanese Encephalitis. Common manifestations are febrile illnesses,
encephalitis, hemorrhaging, and hepatitis. Most of theses viruses are
transmitted by mosquitoes. Hepatitis C, discovered in 1989, is
transmitted by blood contact like other Hepatitis viruses, and it is as
important as Hepatitis A and B.) recombine with a wild-type virus, a new microbe
with potentially undesirable properties could result, according to
Stephen J. Seligman, M.D., research professor of microbiology and
immunology. His paper, "Live flavivirus vaccines: reasons for caution"
appears as a rapid review article in the June 19, 2004, issue of the
journal Lancet. Co-author is Ernest A. Gould, Ph.D., of CEH-Oxford in the
UK.
Flaviviruses, which can recombine within species and may recombine
between species, also include dengue, Japanese encephalitis and
tick-borne encephalitis. They cause substantial sickness and death each
year. Although live-virus vaccines offer great promise in terms of cost
and efficacy, their use should be approved by an international authority
due to safety concerns, the authors write.
The article lists five main lessons learned from other live virus
vaccines:
Reversion of vaccine strains to increased virulence;
Development of disease in individuals with compromised immune systems;
Birth defects, particularly if the vaccine is given in the first
trimester;
Spread of vaccine strains to unvaccinated persons and;
The discovery of new, previously undetermined complications.
Bats, Mosquitoes and Dollars
by Dr. Charles A. R. Campbell
San Antonio Texas in 1920 faced a yellow fever epidemic and so many
mosquitoes that one could barely open one's mouth on a summer's night
without getting a mouthful of mosquitoes in it. Dr. Campbell persuaded the
city fathers to build a couple of municipal bat roosts, and in just a few
months, the mosquito problem was gone. Apparently, a single bat can capture
and eat many thousands of mosquitoes in one night. They confirmed this by
measuring the blood iron content of the bat guano left in the roosts. Why
can't we use this method today, instead of spraying dangerous pesticides?
http://www.rawfoodinfo.com/Free%20Stuff/free%20stuff.html
Click
here for the package insert to yellow fever vaccine
Read this from the National Library of medicine.
http://profiles.nlm.nih.gov/LW/Views/Exhibit/narrative/disease.html
"After the U.S. entered the war in late 1941, the army started
vaccinating all troops, not just those headed to tropical areas. By early
April 1942, the IHD had furnished seven million doses to the U.S. Army and
Navy, British forces in Africa, and others. In March, however, many cases of
hepatitis--mostly mild--were being reported in American troops especially in
California. Sawyer and Bauer went out to investigate, and within a month the
evidence pointed to infection from nine specific lots of vaccine. These, in
turn were traced to 2 percent of the blood donors, who had histories of
hepatitis but no symptoms at the time they gave the blood. The level of
production had required a much larger pool of blood donors for the serum,
and no one had checked on their medical histories.
"At Sawyer's direction, the IHD converted to serum-free vaccine production
by June of 1942, but in the end there were still over 49,000 cases of
vaccine-related hepatitis that year, 84 of them fatal."
MTU lowers flags to honor dead grad student
http://www.wlns.com/Global/story.asp?S=4109127
HOUGHTON, Mich. Flags at Michigan Tech in Houghton are flying at half-staff
to honor a 22-year-old grad student who died from a reaction to yellow fever
vaccine. Danielle Ladwig died two weeks ago at the Mayo Clinic in Minnesota.
Ladwig got the inoculation in preparation for a trip to Bolivia to help
build a septic system for a new school. Ladwig was a member of the
group Engineers Without Borders. Her funeral is scheduled tomorrow in her
Wisconsin hometown.
Copyright 2005 Associated Press. All rights reserved. This material may not
be published, broadcast, rewritten, or redistributed.
_____
Malaria Info
meningitis
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